CDR1as contained approximately 74 binding sites for miR‐7, and CDR1as over‐expression down‐regulated miR‐7 expression levels.20, 34 The repression of miR‐7 function by CDR1as revealed a new therapeutic strategy for treatment of Alzheimer's disease. The gene discussed is LINC00632; the disease is early-onset autosomal dominant Alzheimer disease.