In addition, a follow-up study, in which data from the Turkish GWAS were imputed, identified four new loci contributing to the BD susceptibility, CCR1/CCR3 (C-C motif chemokine receptor 1/3), STAT4 (signal transducer and activator of transcription 4), KLRC4 (killer cell lectin like receptor C4), and ERAP1 (endoplasmic reticulum aminopeptidase 1) (19). This evidence concerns the gene CCR1 and Behcet disease.