Erythromycin reduced ribonuclear foci and reversed mis-splicing of several events, including Atp2a1 exon 22 and Mbnl1 exon 5 and 7 in the C2C12 DM1 transgenic cell model at an optimal dose of 50 μM [46]. Here, MBNL1 is linked to myotonic dystrophy type 1.