MYH7 and heart failure: Another key feature of hypertrophy observed in both human heart failure and AngII‐treated mouse cardiomyocytes is the enrichment of foetal‐like gene programmes involved in cardiac remodelling 47, including upregulation of hypertrophic markers such as Myh7. Importantly, additional treatment with ldCil restored Myh7 levels back to untreated levels (supplementary material, Figure S5).