In the more recent review article [5], Chang states that “DIC [disseminated intravascular coagulation] has been inappropriately conceptualized as a fibrin clot disease produced via activated TF/FVIIa-initiated cascade/cell-based coagulation” and that “consumption coagulopathy in acute promyelocytic leukaemia that occurs due to pathologic activation of aberrant TF path caused by TF released from leukemic promyelocytes should be called true DIC [8]”. Here, TF is linked to glycogen storage disease VI.