ARG1 was shown to contribute to asthma pathogenesis by inhibiting nitric oxide production, modulating fibrosis, regulating arginine metabolism and inhibiting T cell proliferation, and it involves the initiation of adaptive T helper 2 cell‐mediated allergic lung inflammation by regulating group 2 innate lymphoid cells 57, 58, 59, 60, whereas ARG1 ablation in the lung may enhance peripheral lung function but have little effect on airway inflammation 61. Here, ARG1 is linked to inflammatory response.