NFKB1 and inflammatory bowel disease: To explore whether MCT4 contributed to IL‐6 expression by promotion of NF‐κB activity, we found that the relative abundance of NF‐κB p65 in nucleus was decreased to 25% of control group by CHC treatment (Figure 5E‐F), and NF‐κB p65 in nucleus was evaluated in colonic epithelial of patient with IBD compared with healthy donors (Figure 5G).