Regarding the circumstance of a tumor that evades growth suppressors, it could be useful to cite Nahta et al. [69], who showed that bisphenol A, a common component of plastic which exerts estrogenic effects, promotes cell cycle progression by disrupting multiple targets, including functional p53, determining dose-dependent evasion of apoptosis and increased proliferation [69,70]. This evidence concerns the gene TP53 and neoplasm.