Consistent with our results using WT C57BL/6J mice (Fig 1), infection had no influence on BALF IL-11 concentrations, nor did NFκB targeting in either cell type (Fig 3), suggesting that the relatively high abundance of this cytokine is maintained by alternative signaling requirements and/or cell types other than those of myeloid or epithelial lineage. The gene discussed is NFKB1; the disease is infection.