However, composite assessment of GFAP and UCH-L1 (in addition to spectrin break down product (SBDP-150) revealed that these proteins were elevated in blood collected from study participants who had the most striking decrements in neurocognitive performance, including simple reaction time, as well as symptom reporting [19] in the absence of a clinically defined concussion. The gene discussed is GFAP; the disease is concussion.