hAFSCs have the potential to migrate to the lesion via CXCL12/CXCR4 signaling, based on the results from several rodent models such as bleomycin‐induced pulmonary fibrosis 29, renal intestinal fibrosis 30, and a sciatic nerve injury 31; thus, in our study, hAFSCs injected into the uterine cavity might accumulate in the lesion due to concentration gradients of CXCL12 that originate from the exposed spinal cord. This evidence concerns the gene CXCR4 and pulmonary fibrosis.