CXCL12 and pulmonary fibrosis: hAFSCs have the potential to migrate to the lesion via CXCL12/CXCR4 signaling, based on the results from several rodent models such as bleomycin‐induced pulmonary fibrosis 29, renal intestinal fibrosis 30, and a sciatic nerve injury 31; thus, in our study, hAFSCs injected into the uterine cavity might accumulate in the lesion due to concentration gradients of CXCL12 that originate from the exposed spinal cord.