Consistent with this notion, the combination of BET bromodomain inhibitor JQ1 and TKIs has been suggested to be a rational strategy for treating leukemia and lymphoma.42 Additionally, BRD2 was shown to positively control epithelial‐mesenchymal transition in breast cancer,43 and a GS for this histologic transformation could forecast the resistance to EGFR inhibitor erlotinib in both wild‐type EGFR and mutant EGFR lung cancer cases.44 This evidence concerns the gene DNER and lung cancer.