Given the pivotal role of Janus kinase (JAK) activation in tumor‐promoting inflammation and its oncogenic functions, JAKs represent attractive therapeutic targets in tumorigenesis.8 The JAK family is composed of four members (JAK1, JAK2, JAK3 and tyrosine kinase 2) and couples extracellular stimuli with gene transcription. The gene discussed is JAK2; the disease is neoplasm.