CAMP and psoriasis: Finally, the more recent discoveries that complexes of the antimicrobial peptide LL37 (a 37 amino acid C-terminal cleavage product of the antimicrobial peptide, cathelicidin) with own DNA or the melanocytic antigen ADAMTSL5 may function as autoantigens (27, 28), support the central role of T cells in the pathogenesis of psoriasis (29, 30).