FFAs induce breast cancer invasion by activating the epidermal growth factor receptor, GTP-binding protein, and protein kinase C pathway [57], and by controlling cell proliferation via phosphatidylinositol 3-kinase (PI3K) [58] and cell migration through free fatty acid receptor 1 and 4 and AKT pathway activation [59]. The gene discussed is AKT1; the disease is breast carcinoma.