A conserved role for LIN‐53 in aging regulation is conceivable because its human homologs RBBP4 and RBBP7 have been implicated in Hutchinson–Gilford progeria syndrome (HGPS), which leads to premature aging (Pegoraro et al., 2009). The gene discussed is RBBP7; the disease is Hutchinson-Gilford progeria syndrome.