ICAM1 and diabetes mellitus: Interestingly, both in in vitro and in vivo models of diabetes, the overexpression of endothelial adhesion molecules (ICAM-1, VCAM-1, Selectins), and of the enzymes implicated in their O-GlcNAcylation induced by high glucose levels, were significantly decreased by miR-200a and miR-200b mimics (Lo et al., 2018).