Recent data suggested that miR-361 directly targeted the 3′-UTR of FGFR1, which promotes glycolysis through activation of two critical glycolytic enzymes lactate dehydrogenase A (LDHA) and pyruvate dehydrogenase kinase 1 (PDK1/PDHK1), thereby suppressing glucose consumption and lactate production of breast cancer cells [19]. This evidence concerns the gene LDHA and breast carcinoma.