The role of miR‐29a in glucose metabolism was observed not only in adipocytes but also in skeletal muscle cells, as the overexpression of miR‐29a led to insulin resistance by inhibiting the proliferator‐activated receptor δ(PPARδ), and peroxisome proliferator‐activated receptor‐γ coactivator‐1α (PGC‐1α) signals and GLUT4, which are responsible for insulin‐stimulated glucose uptake (Zhou, Gu, et al., 2016). This evidence concerns the gene SLC2A4 and Insulin resistance.