VWF and kidney injury: In experimental models of kidney injury, prolongation in buccal mucosal bleeding time (BMBT) and decreased platelet glass bead retention percentage occurs without significant changes in arachidonic acid (AA)‐, adenosine diphosphate (ADP)‐, collagen (COL)‐, and epinephrine‐induced light transmittance aggregometry (LTA).10 In addition, vWF antigen concentrations (vWF:Ag) increases,11 suggesting 1 of the mechanisms of bleeding to be due to abnormalities in platelet adhesion independent of vWF or platelet agonists.