Increased numbers of γδT17 cells were found in the livers of high-fat diet (HFD)-fed mice and their NAFLD symptoms were reduced in Tcrδ−/− mice, suggesting that hepatic resident γδT17 cells are one of the main sources of IL-17A in the liver during NAFLD and can accelerate NAFLD progression (16). Here, IL17A is linked to metabolic dysfunction-associated steatotic liver disease.