ACE2 and autoimmune uveitis: Therefore, it appears that reduction of AngII expression (for instance, by blocking prorenin or renin actions) or blockade of AT1R, on the one hand, and stimulation of the ACE2/Ang (1–7)/Mas axis, on the other, may be exploited to counteract retinal damage occurring in retinal pathologies such as glaucoma, retinal ischemia, autoimmune uveitis, or diabetic retinopathy.