CD200R1 and fungal infectious disease: Eosinophils, neutrophils and CD4+ T cells were devoid of CD200R1 at homeostasis and upon C. neoformans infection (Fig. 1C), suggesting that any impact of CD200R1 manipulation in the context of fungal infection was likely mediated through macrophages (refer to Supplementary Fig. 1 for flow cytometry gating strategy).