Indeed, neutrophils could contribute to parasitemia control by (a) phagocytosis, (b) granular secretion of antibacterial compounds, (c) release of neutrophil extracellular traps (NETs), and (d) the induction of a hostile inflammatory environment.22, 23, 24, 25, 26 The latter, that is the combined action of neutrophil‐derived tumour necrosis factor (TNF) and NO, could aid the significantly weakened remaining antibody response in maintaining a certain parasitemia control lever during later stages of infection. Here, TNF is linked to parasitic infectious disease.