PHB2 and myocardial infarction: In the former, Wang et al. showed that, in MI mice, the cardiac apoptosis-related lncRNA (Carl) can suppress apoptosis and mitochondrial fission in anoxic cardiomyocytes by acting as a ‘sponge’ for miR-539, which in turn targets prohibitin-2 (Phb2), a gene important for mitochondrial function [32].