Notably, an estimated 60–69% of triple negative breast cancers (with absence of oestrogen receptor (ER) progesterone receptor (PR) as well as nonamplified HER2) are reported to have a defect in DNA repair, with features in common with BRCA1/2 mutated tumors described as “BRCAness” [5, 6]. Here, PGR is linked to triple-negative breast carcinoma.