There was no modulation observable due to either RSVA2 or RV2 infection compared to untreated controls (lane 5 vs lane 1) or even poly(I:C) stimulation (lane 2 vs lane 1 at 1h; lane 6 vs lane 5 at 4 h), suggesting that translation of IFN genes in AECs is not dependent on phosphorylation of eIF4E. Here, IFNA1 is linked to infection.