Similar to our study, recent studies have shown that MAP3K1 is functionally required for multiple physiological processes, including cell growth, cell migration, and apoptosis, and is strongly correlated with poor outcomes in a wide range of malignant cancers.13, 14, 15 Furthermore, another study demonstrated that MAP3K1 promotes tumorigenesis in nonsmall cell lung cancer cells by regulating epithelial‐mesenchymal transition through miR‐145‐5p‐mediated JNK signaling pathway activation, suggesting the potential role of MAP3K1 as an important oncogene.12, 15, 35. The gene discussed is MAP3K1; the disease is lung cancer.