From the total of 149 GCMs identified in the genome-wide coexpression analysis, we found that 74 GCMs (49.66%) include genes that are differentially expressed between responders and non-responders to anti-TNF therapy, 9 GCMs (6.05%) include genetic variation underlying anti-TNF response, and 37 GCMs (24.83%) include susceptibility genes for RA (Table S3). Here, TNF is linked to rheumatoid arthritis.