TGM2 and myeloid sarcoma: Of note, in contrast to astrocytes in the cuprizone model (Skripuletz et al., 2013; Tameh et al., 2013) and in MS lesions, (Williams et al., 2007; Gudi et al., 2014; Lassmann, 2014), the hTG2-expressing astrocytes used in the present study were not subjected to an inflammatory environment which could have implications for their function.