A recent meta-analysis showed a significant difference in MMP-9, tissue inhibitor of matrix metalloproteinase 1 (TIMP-1), and CRP between patients with and without AAA [15]; MMP-3 and MMP-9 levels were higher in patients with CAE than in patients with coronary artery disease and normal coronary arteries.[16] In the present study, there were no significant differences in MMPs in patients with/without AAA and with/without CAE. The gene discussed is CRP; the disease is triple-A syndrome.