Two recent studies found similar antibody reactivity against group A CIDRα1 in uncomplicated and severe malaria during acute disease (34, 35), while at convalescence older children with severe (likely noncerebral) malaria had higher antibody levels against such EPCR binding CIDRα1 than those with uncomplicated malaria (34). The gene discussed is PROCR; the disease is glycogen storage disease VI.