C1QTNF3 and myocardial infarction: In addition, we observed that CTRP3 overexpression via infusion of CTRP3-engineered MSCs dramatically decreased vimentin positive cells (Fig. 3c, f) and enhanced the effects of MSCs on inhibition of myocardial apoptosis in the border zone 28 days after MI (Fig. 3g, h).