The presence of somatic mutations in genes including NPM1 (Nucleophosmin 1), CEBPA (CCAAT/enhancer binding protein alpha), c-KIT (tyrosine-protein kinase Kit) and FLT3 (Fms-like tyrosine kinase 3) can also promote myeloid leukemogenesis and influence the prognosis of AML [3]. This evidence concerns the gene KIT and acute myeloid leukemia.