AKT1 and gestational diabetes: MicroRNA‐351 is a family member of the interferon β (INFβ)‐inducible mRNAs which plays a role in accelerating cellular antiviral activities.19 MicroRNA‐351 level is temporarily elevated in the period of muscle regeneration and implicated in muscle atrophy.20 Based on the aforementioned literature, we could suggest that miR‐351 regulated GDM by mediating FLOT‐dependent PI3K/AKT pathway.