The results of Andrukhova et al., showing elevated GSTP1 expression in the failing myocardium, were associated with reduced GSTP1:JNK interaction and consequent activation of the JNK-MAPK signaling cascade, essential for cardiomyocyte apoptosis [37], representing further confirmation of the contributing role of oxidative stress in the HF progression. Here, GSTP1 is linked to hydrops fetalis.