A subset of patients infected with H. pylori have been found to have elevated levels of autoantibodies against proteins essential for normal neurological functions (Nuclear factor 1 A-type (NFIA), Platelet Derived Growth Factor Subunit B (PDGFB), eukaryotic translation initiation factor 4A3 (EIFA3), suggesting a mechanism of molecular mimicry contributing to increased PD motor severity in H. pylori seropositive individuals (77). The gene discussed is NFIA; the disease is Parkinson disease.