It may thus coordinate valence surveillance, which integrates stimuli of different modalities and priorities to generate a specific purposive behavior.165 For instance, stimulation of beta‐adrenoreceptors in the ventral BNST has been shown to reduce food intake while increasing anxiety.169 On the other hand, recent evidence demonstrates that NPY acting on vBNST Y2R reduces fear expression and promotes fear extinction.63, 64 Interestingly, the activation of vBNST Y2R was able to induce long‐term suppression of conditioned fear even in the absence of extinction training. The gene discussed is NPY; the disease is Anxiety.