CYTOR and gastric cancer: When we investigated the relationship between linc00152 and gastric carcinogenesis, we observed that linc00152 can specifically recognize the EGFR‐binding site and activate the PI3K/AKT signaling pathway to promote the proliferation of gastric cancer cells.31 Moreover, PI3K signals help regulate many cellular functions such as proliferation, differentiation, apoptosis, and glucose transport.