Since we previously showed increased endothelial expression of CathL in the omenta of patients with metastasised serous carcinoma [41], the current observation of a similar increased Gal1 expression pattern in comparable patient samples could signify a physiological role for CathL induction of Gal1 expression in angiogenesis in this pro-metastatic disease. This evidence concerns the gene LGALS1 and serous adenocarcinoma.