Interferon gamma (IFNγ) plays a pivotal role in antimicrobial defense, and loss‐of‐function mutations in IFNγ‐receptor signaling in the human and mouse result in severe susceptibility to infections, including by bacteria and microbial parasites (Altare et al, 1998; Levin & Newport, 1999; Janssen et al, 2002). This evidence concerns the gene IFNG and infection.