DYRK1A and Dravet syndrome: Mice overexpressing DYRK1A have increased pTau in the brain, while crossing DYRK1A heterozygous mice with DS mice regulated pTau, amyloid load, and symptoms (García‐Cerro, Rueda, Vidal, Lantigua, & Martínez‐Cué, 2017), implying DYRK1A is a highly dosage‐sensitive gene.