SELP and acute coronary syndrome: These possible roles of P-selectin in atherogenesis are further underscored by its higher surface expression on platelets in acute coronary syndromes other than stable angina10, and mitigation of myocardial injury by the P-selectin antagonist inclacumab in patients with ST-segment elevation myocardial infarction (STEMI) undergoing percutaneous coronary intervention (PCI)11.