Potential causes of inflammatory arthritis and RA are thought to involve both genetic and environmental factors [2, 3], and its pathogenesis involves the high production of proinflammatory cytokines, such as tumor narcosis factor (TNF), interleukin 1 beta (IL1B), interleukin 6 (IL6), and interleukin 17A (IL17A), inducing autoimmune activity, thereby inflaming the synovial membrane [4]. This evidence concerns the gene IL1B and rheumatoid arthritis.