In silico analysis reveals pathways underlying the aberrant proliferation and survival of nascent lymphoma cells, and identifies genes likely to be involved in the subversion of failsafe processes that precede rapid onset lymphomagenesis.18 As MYC and RUNX overexpression have been implicated in a wide range of malignancies, these observations may be of wide relevance to cancer.9 Here, MYC is linked to lymphoma.