NR1H2 and adrenal cortex carcinoma: Inhibition of HDAC5 by SAHA in cultured neurons led to the induction of AcH3 and Lxrβ (Fig. 7c–e), and ChIP data showed a significant binding of histone acetylation to the promoters of Lxrβ (Fig. 8), indicating that LXRβ was a downstream target of HDAC5 in ACC of CIP mice.