The development of CNV is subsequently followed by subretinal fibrosis, which accounts for most cases of the severe vision loss in the late stage of AMD, leading to permanent photoreceptor damage and vision loss.2, 3 Among the several molecular players involved in the pathogenesis of CNV, vascular endothelial growth factor (VEGF)-A plays distinct roles in pathological angiogenesis and inflammation in AMD.4 Here, VEGFA is linked to age-related macular degeneration.