To explore this possibility, our gene ontology analysis (Table 3) revealed that most of the DMLs we observed that relate to insulin resistance occured at genes involved in aerobic respiration (NDUFS7), growth hormone secretion (RAB1A), positive regulation of macrophage chemotaxis (CMKLR1), and rapid glucocorticoid signaling (MAPK11). The gene discussed is GH1; the disease is Insulin resistance.