Angiomotin like 1 (AMOTL1), that promotes tube formation and migration of endothelial cells and regulates tight junctions, cell polarity, and epithelial-mesenchymal transition in epithelial cells [41], and Von Hippel-Lindau (VHL), a tumor suppressor involved in the ubiquitination and degradation of hypoxia-inducible-factor, are verified miR-23b target genes [42, 43]. This evidence concerns the gene VHL and neoplasm.