While BACE1-AS, NATRad18,17A, and hnRNP Q lnRNAs have been found to be associated with Alzheimer's disease, in this research study the significance of themost known β-turn-forming residues between these proteins is computationally identified and discussed, as a potentially crucial factor onthe regulation of folding, aggregation and other intermolecular interactions. This evidence concerns the gene SYNCRIP and early-onset autosomal dominant Alzheimer disease.